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Defect In Brain Causes Erectile Dysfunction In Diabetics

Erectile dysfunction (ED) in diabetics is due to a selective defect in the brain, according to a study conducted by researchers at the University of Nebraska Medical Center.

It was observed that erectile dysfunction in diabetics was due to a loss in the synthetic enzyme for the production of nitrogen oxide (NO) within the neurons of the PVN.

The findings, which were published in the American Journal of Physiology, said the defect is a loss in the synthetic enzyme for the production of nitrogen oxide (NO) within the neurones of the paraventricular nucleus (PVN) of the hypothalamus, located in the brain, an integration center between the central and peripheral nervous systems.

According to the researchers restoring this synthetic enzyme may have a significant effect for diabetic patients with erectile dysfunction.

During the study, the researchers examined stimulated penile erection.

They focused on the paraventricular nucleus (PVN), which is involved in various functions including erectile function and sexual behaviors. Researchers further examined central nitric oxide that plays major role in the neurotransmission of normal penile erection.

Penile erection is a behavioral response that occurs in response to the administration of N-methyl-D-aspartic acid (NMDA) within the PVN. At the same time, inhibition of NO synthase with NG-monomethly-L-argining (L-NMMA) prevents NMDA-induced erection.